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In the study (Budde et al, 2000; Jørgensen and Gudbjerg, 2003), it was observed that 5‐HT2A antagonists had an antidepressant effect on rats who were treated with diazepam, an increase in the forced swim test, and decreased immobility in the forced swimming test when compared with saline (Budde et al, 2000). Also, 5‐HT2A agonists reduced immobility in the forced swim test and latency to the immobility test (Jørgensen and Gudbjerg, 2003).
The study of Kivisild et al. (2000) showed that 5‐HT2A antagonists were able to enhance the antidepressant activities of amfonelidine. This study also showed that 5‐HT2A agonists enhanced the antidepressant effects of amfonelidine.
The study (Lundstrom et al, 2001) showed that 5‐HT2A agonists were able to decrease the immobility time during forced swim test.
In the study (Till et al, 2002), it was observed that 5‐HT2A antagonists were able to attenuate immobility and reduce the rate when compared with saline treatment rats were given repeated injection of amfonelidine.
In the study (Watkins et al, 2006), it was observed that 5‐HT2A antagonists in the low dose used produced a significant reduction in immobility time.
In the study (Ridker et al, 2006), 5‐HT2A agonists in the low dose used produced a significant reduced immobility time.
The study (Munro et al, 2007), however, showed that 5‐HT2A agonists produced little or no antidepressant‐like activity in the forced swim test.
Also, the study (Gill and Jones, 2008), in which 5‐HT2A antagonists but not 5‐HT2C agonists were used as monotherapy, showed that a decrease in immobility time was evident after 6 weeks of treatment, and that there was no difference between groups of amfonelidine recipients, in terms changes the immobility time.
The study (Rehding et al, 2007), in which 5‐HT2A agonists but not 5‐HT2C were used as monotherapy, showed that a decrease in immobility time was evident after 6 weeks of treatment, and that there was no difference between groups of amfonelidine recipients, in terms changes the immobility time.
Another study (Sørensen et al, 2008) showed that a decrease in immobility time was evident after 15 days of treatment with 5‐HT2A agonists and 5‐HT2C agonists, which was followed by an increase in immobility time.
The study (Berthelsen et al, 2008) showed that a decrease in immobility time was evident after 6 weeks of treatment with 5‐HT1A agonists and 5‐HT2C anxiolytics.
In the study (Berthelsen et al, 2008), which was conducted in healthy men and women, it was observed that an increase in immobility time was evident the treated group after 6 weeks of treatment with 5‐HT1A agonists, and that this increase was not due to a different dose‐response pattern.
In the study (Berthelsen et al, 2008), which was conducted in healthy men and women, it was observed that an decrease in immobility time was evident after 8 weeks of treatment with 5‐HT1A agonists, and that this decrease was not due generic drugstore makati to a different dose‐response pattern.
In the study (Berthelsen et al, 2008), in healthy men and women, it was observed that an increase in immobility time was evident the treated group after 9 and 10 weeks of treatment with 5‐HT1A agonists and 5‐HT1C agonists, respectively, that this was prevented by a 6‐week washout.
In the study (Berthelsen et al, 2008), which was conducted in healthy men and women, which five doses of 5‐HT1A agonists and five doses of 5‐HT1C agonists were administered in single doses, a decrease immobility time was evident after 6 and 8 weeks of treatment, as was a decrease in the number and duration of events immobility assessed.
In the study (Berthelsen et al, 2008), which was conducted in healthy men and women, five dosages of 5‐HT1A agonists was administered in single doses, a decrease immobility was evident after 6 and 8 weeks of treatment, as was a decrease in the number and duration of.
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